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Kyle Jansson, MD, PhD

Kyle Jansson portrait
Assistant Professor, The University of Kansas Medical Center, Nephrology and Hypertension
kjansson@kumc.edu

Professional Background

Assistant Professor of Medicine
Department of Internal Medicine, Division of Nephrology and Hypertension

MD - KUMC
PhD - KUMC
Residency in Internal Medicine - KUMC
Fellowship in Nephrology and Research - KUMC

Clinical Interests:
My clinical interests are in the areas of chronic kidney disease and polycystic kidney disease.

Education and Training
  • MD, Medicine, The University of Kansas Medical Center, Kansas City, KS
  • PhD, Molecular and Integrative Physiology, The University of Kansas Medical Center, Kansas City, KS
  • Internship, Internal Medicine, The University of Kansas Medical Center, Kansas City, KS
  • Residency, Internal Medicine, The University of Kansas Medical Center, Kansas City, KS
  • Other, Internal Medicine, The University of Kansas Medical Center, Kansas City, Kansas
  • Other, Nephrology, The University of Kansas Medical Center, Kansas City, KS
Licensure, Accreditations & Certifications
  • Internal Medicine, American Board of Internal Medicine
  • Medical License, Kansas State Board of Healing Arts
  • Nephrology, American Board of Internal Medicine

Research

Overview

Research Interests:
I am a researcher in the Jared Grantham Kidney Institute at the University of Kansas Health System. My research interest is in the role of phosphaturia in mechanisms of kidney injury and inflammation in CKD.

Publications
  • Stubbs, Jason., R, Zhang, Shiqin, Jansson, Kyle., P, Fields, Timothy, Boulanger , Liu , Rowe, Peter., S. 2022. Critical Role of Osteopontin in Maintaining Urinary Phosphate Solubility in CKD. Kidney360
  • Jansson, Kyle., P, Yu, Alan, Stubbs, Jason., R. 2022. Contribution of Phosphate and FGF23 to CKD Progression. Current Opinions Nephrology and Hypertension, 31 (4), 306-311
  • Jansson, Kyle., P, Vanugopal , Magenheimer , Reif , Wallace , Calvet , Blanco . 2015. Ouabain regulates CFTR-mediated anion secretion and Na,K-ATPase transport in ADPKD cells. Journal of Membrane Biology, 248
  • Jansson, Kyle., P, Magenheimer , Maser , Calvet , Blanco . 2013. Overexpression of the polycystin-1 C-tail enhances sensitivity of M-1 cells to ouabain. Journal of Membrane Biology, 246 (7), 581-590
  • Jansson, Kyle., P, Nguyen , Aramadhaka , Reif , Magenheimer , Bello-Reuss , Wallace , Calvet , Blanco . 2012. Endogenous concentrations of ouabain act as a cofactor to stimulate fluid secretion and cyst growth of in vitro ADPKD models via the cAMP and EGFR-Src-MEK pathway. American Journal of Physiology Renal Physiology, 303 (7), 982-990
  • Nguyen, Anh, Jansson, Kyle., P, Sanchez , Sharma , Reif , Wallace , Blanco . 2011. Ouabain activates the Na,K-ATPase signalosome to induce autosomal dominant polycystic kidney disease cell proliferation. American Journal of Physiology Renal Physiology, 301 (4), 897-906