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Katie L. Dennis, MD

Katie Dennis portrait
Clinical Associate Professor, Pathology and Laboratory Medicine

Professional Background

Dr. Katie Dennis is an Associate Professor in the Department of Pathology and Laboratory Medicine. Dr. Dennis subspecialties include but are not limited to the following: Cytopathology, liver pathology, autopsy, gastrointestinal/pancreatic pathology, breast pathology and genitourinary pathology.

Education and Training
  • Clinical Fellowship, Post-sophomore fellowship Pathology, University of Kansas Medical Center
  • Residency, Pathology, University of Kansas Medical Center
  • Clinical Fellowship, Cytopathology Fellow, University of Kansas Medical Center
Professional Affiliations
  • American Society for Clinical Pathology, Member, 2009 - Present
  • College of American Pathologists, Member, 2004 - Present
  • Kansas City Society of Pathologists, Member, 2004 - Present
  • United States and Canadian Academy of Pathology, Ad-Hoc Member, 2004 - Present



Dr. Dennis research interests are in the following areas: genitourinary pathology, liver pathology, cytopathology and resident education. Dr. Dennis is actively involved in the teaching of medical students, pathology residents, surgical pathology and cytopathology fellows.

  • Schloegel , Van, Alam, S, Dennis, Katie, Holzbeierlein, J, Taylor, John A III. 2020. Near Miss Diagnoses of Solitary Bladder Tumors Highlight the Importance of Immunohistochemical Staining.
  • Mettman, Daniel , Haer, E, Olyaee, M, Rastogi, A, Madan, R, O'Neil, M, Kelting, S, Dennis, Katie. 2020. The utility of immunohistochemical testing for mismatch repair proteins in fine needle aspiration specimens of pancreatic adenocarcinoma
  • Plummer, RM, Kelting, S, Madan, R, O'Neil, M, Dennis, K, Fan, F. 2020. Triaging of pleural effusion cytology specimens for ancillary flow cytometric analysis
  • Dennis, Katie, Fang, Fan. 2020. Ch 7: Lung and Respiratory Tract Cytology, 95-142
  • Dennis, Katie , Wang, Y, Li, J, Zhang , Y. 2018. Bile acids regulate cysteine catabolism and glutathione regeneration to modulate hepatic sensitivity to oxidative injury.