Former Members

Postdoctoral Fellows
Martha Carletti, PhD Martha Carletti, PhD: Martha joined the Behbod laboratory after successfully defending her dissertation entitled "MicroRNA-21 Mediated Post-Transcriptional Gene Regulation in Ovarian Function" July 10, 2009 through the University of Kansas School of Medicine. Her research association in the laboratory resulted in co-authorship for manuscript publications in Breast Cancer Research and Current Drug Targets during 2010 and 2011. 
Arindam Paul, PhD Arindam Paul, PhD: Arindam was in the Behbod lab from August of 2009 through May of 2011. His research focus was in understanding the molecular mechanism for DCIS to Invasive Breast Cancer (IBC) progression. He also researched understanding the role of p63 during DCIS to IBC progression.
Research Assistants  
Kevin Campbell, BS:  Kevin was a member of the Behbod laboratory from August, 2009 through June, 2010. He spent his year in the lab as a Research Associate. His main responsibilities included formulating, planning and carrying out individual experiments and research, purchasing and stocking of all lab materials, assisting four different PhD researchers in their experiments and maintenance of animal models. Kevin is currently in his third year as a medical student at the University of Kansas School of Medicine.
Sofia Kerbawy, MD: Sofia was a member of the Behbod laboratory from Summer, 2007 through July, 2009, when she joined the MD program at the University of Kansas School of Medicine. While in the laboratory she served as a Research Assistant and was a co-author on two manuscripts appearing Breast Cancer Research. She is currently a pediatrics resident at Children's Hospital and Research Center, Oakland, California.
Daniel Newman Daniel Newman: Daniel was a member of the Behbod laboratory from 2012 through the spring of 2013. The primary goal of Daniel's research utilizes novel imaging technology, Raman microspectroscopy (RM) in combination with flow cytometry to allow for accurate detection and characterization of cells with distinct intracellular molecular profiles (IMPs) in order to discern heretogeneous cells among an otherwise homogeneous population of tumor-initiating cells (TICs; Lin-CD24hiCD29hi). By performing this study, it is anticipated that future genomic and proteomic characterization of cells with distinct Raman profiles may be used to discover signaling pathways or genes contributing to increased tumorigenic capacity.
Medical Students
Adrianne Eyman, BA Adrianne Eyman, BA: Adrianne worked in the lab during the summer of 2013.  She studied radioresistance in DCIS, specifically the role of canonical Wnt Pathway in promotion of cellular radioresistance. Adrianne is currently in her second year as a medical student at the University of Kansas School of Medicine.
Emily Steffensen Emily Steffensen:Emily is a KINBRE scholarship recipient (April 2014 - May 2015) enrolled in pre-medicine biology and physics, currently on a research rotation. Emily's project involves testing the effectiveness of carnosic acid (CA) as a potential treatment for preventing non-invasive forms of breast cancer from becoming invasive. She is performing various experiments to assess effects of CA on cell proliferation as well as migration and invasion. Further studies analyzing CA's effect on transcriptional signaling via the Wnt pathway will also be executed; then she will be able to test CA in an in vivo mouse intraductal model that mimics the process of non-invasive ductal carcinoma in situ (DCIS) progression to invasive ductal carcinoma (DC).
Aria Sabbagh Aria Sabbagh: Aria is a KINBRE scholarship recipient (April 2014 - May 2015) enrolled in pre-medicine, currently on a research rotation. The purpose of Aria's project is to determine the role of nuclear BCL9 in DCIS invasive progression. He is working on finding the nuclear localization signal of BCL9 in order to delete this sequence using CRISPR genomic editing. By deleting the nuclear localization signal, he will be able to determine the cytoplasmic functions of BCL9 in mouse xenograft models of DCIS. The hypothesis is that if BCL9 is blocked from entering the nucleus, then proliferation, invasion, and angiogenesis will be significantly decreased in DCIS xenograft models.
Shadow Student
Linzi Oppenheimer Linzi Oppenheimer: Linzi volunteered in Dr. Behbod's laboratory from June 3 to August 2, 2013 and again during the summer of 2014. She is currently a student at Blue Valley North High School. She helped in all projects in the lab. Specifically, she worked on experiments to test the role of canonical Wnt pathway in promoting DCIS cell radio-resistance. Linzi is interested in becoming a medical doctor in the future.


Last modified: Oct 31, 2014